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Glucagon-like peptide-1 decreases endogenous amyloid- peptide (A) levels and protects hippocampal neurons from death induced by A and iron
EV: Extracellular vesicle
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A series of secondary injury cascades, such as the release of blood metabolites, microglial activation, thrombin activity, the release of excitatory amino acids, and proinflammatory factors, are initiated (Figure 1)
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These changes indicate the peptide is active even before hair changes become apparent
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