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They are pharmacologically active compounds with CNS effects that current safety databases have not systematically catalogued
Notably, Cagrilintide activated fewer neurons overall than sCT, particularly in the AP by 75% and LPBN by 36%, suggesting differences in their receptor selectivity and downstream signaling dynamics [2]
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Secondary antibodies, diluted 1:200, were incubated in 0.05% tween-20 in PBS for 1 h at 37 C
NIASPAN
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