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Studies in ovariectomized mice showed that estrogen replacement increased GLP-1 release from both intestinal L-cells and pancreatic alpha-cells

The effects of norepinephrine are exerted through interaction with -adrenergic receptors

Depending on the availability of cellular GSH and the activation of glutathione S-transferase subclasses, detoxification of AFB1 is facilitated [24]

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N-methylated glutathione analogues demonstrate a 16.1-fold increase in oral bioavailability compared to native GSH