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Mukherjee A, Huber K, Evans H, Lakhani N, Martin S

Functional and phenotypic analyses confirm the induction of a pancreatic endocrine fate, including positive dithizone (DTZ) staining indicative of insulin production, as well as the upregulation of key pancreatic markers such as Nestin , PDX-1 , paired box gene 4 ( PAX4 ), neurogenin 3 ( NGN3 ), Isl-1, MAFA, insulin, and glucose transporter 2 ( GLUT2 )

In combination, these drugs promote satiety via enhancement of hypothalamic pro-opiomelanocortin (POMC) cell-mediated release of melanocyte-stimulating hormone (MSH) leading to reduced food intake and increased energy expenditure [16]

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