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J Biol Chem (2000) 275(49):381358
However, studies have shown that antioxidant approaches, including anti-neuroinflammatory and neuroprotective agents, may have therapeutic efficacy in the treatment of PD ( The interplay between mitochondrial function and GSH is especially critical in PD
As well, KYNA-induced inhibition of 7nAChRs reduces presynaptic glutamate release, thereby indirectly dampening AMPAR activation, a mechanism that may protect neurons from glutamate-driven excitotoxicity under pathological conditions [82]
The good, the bad, and the ugly of ROS: New insights on aging and aging-related diseases from eukaryotic and prokaryotic model organisms
Ecotoxicol Environ Saf
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