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Research using the Broad Institute Connectivity Map analyzed 1,309 bioactive molecules and identified GHK-Cu as having remarkable gene expression modulation capabilities

The non-enzymatic mitochondrial protein hyperacetylation induced by the increased availability of mitochondrial acetyl-CoA avoids the risk associated with mitochondria overfeeding, restraining the activity of the respiratory complex I and modulating CACT activity

BPC-157 remains investigational with limited human clinical data

It is not intended for human consumption, medical use, or therapeutic application