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492 However, during sepsis, neutrophils with an extended lifespan and impaired migration are confined to the vasculature, generating overwhelming vascular inflammation through the release of pro-inflammatory cytokines, ROS, and NETs, 493 which eventually results in endothelial dysfunction (Fig

Recent reviews have concentrated on the understanding of oxidative stress and mitochondrial dysfunction in the development of SNHL (Chattaraj et al

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Moreover, when the research was conducted on isolated bovine chondrocytes, it appeared that there might be an increased production of proteoglycan and collagen

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