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[7] The metabolism of cancer cells is also different from the metabolism of normal cells, and these differences can be used to find targets for therapeutic intervention in cancer

IL-22 pretreatment significantly upregulates hepatic LC3-II(the PE-conjugated form of light chain 3) and phosphorylation of AMP-activated kinase (p-AMPK) in APAP-treated mice, and IL-22-mediated LC3-II conversion and protection against APAP-induced cytotoxicity are impaired when p-AMPK is blocked by compound C (an AMPK inhibitor) [203]

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These formats are usually pharmacy-prepared

Given the emerging therapeutic potential of barrier modulating agents, we anticipate that the ability to perturb multiple intestinal tissues and to track subsequent gut barrier functions will further advance mechanistic insights and translational discoveries