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GHK-Cu and BPC-157 could theoretically complement each other in wound healing contexts: GHK-Cu providing copper for extracellular matrix cross-linking and remodeling, while BPC-157 modulates angiogenesis and growth factor signaling

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There is anecdotal support for this approach

Complement activation, cytotoxicity, and alterations in cell junction proteins (e.g., reduced connexin-43) may also exacerbate tissue injury(Wroska et al., 2024)

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