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Blocking GSH synthesis in combination with JMF4073 depletes dTTP/dCTP pools to eradicate T315I-Bcr-Abl leukemia We hypothesize that reducing the GSH level to increase replication stress can render T315I-Bcr-Abl leukemia more susceptible to JMF4073 in vivo

Nonalcoholic fatty liver disease (NAFLD) affects about 25% of the population

Identify the effects of elevated plasma homocysteine on chronic disease risk

TB-500 is studied for actin sequestration, cytoskeletal dynamics, and ILK-AKT cell-survival signaling, which influence cell migration and wound closure