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In high concentrations, this may result in cellular toxicity [3-6] and has the potential to be neurotoxic [7]
Both peptides are derived from human growth hormone, focusing on fat metabolism without impacting IGF-1 levels
Inhibiting GGT impairs GSH ability to restore mTORC1 signaling and ISR, inducing ferroptosis
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Subsequently, we found that the gene and protein expression of IL-17RB in the model group was significantly reversed after SGD treatment
Targeting tumor Gln metabolism has emerged as a prominent focus in tumor metabolic therapy
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