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While more cost-effective than complete media replacement, thermal regeneration may result in some loss of carbon mass

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Known molecular mechanisms driving LEC fibrosis following lens injury/cataract surgery Currently, it is generally accepted that production of fibrotic tissue following lens injury leading to anterior subcapsular cataract, or after cataract surgery to create cells capable of contributing to fibrotic PCO, is driven by transdifferentiation of LECs to myofibroblasts via epithelial to mesenchymal transition (36, 49, 153, 185)

Papaioannou A, Liakopoulou A, Papoulis D, Gianni E, Gkolfi P, Zygouri E