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As defined by BMI, overweight and obesity are highly heritable phenotypes (35, 94)

We interpret the clinical and mechanistic data obtained for different agents that enable weight loss and glucose control for the treatment of obesity and type 2 diabetes mellitus, respectively, by activating or blocking GIPR signalling, including the GIPRGLP1R co-agonist tirzepatide, as well as the GIPR antagonistGLP1R agonist AMG-133

The ECM facilitates immune evasion by forming a physical barrier that hinders immune cell infiltration and impedes the penetration of therapeutic agents [21]

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