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Hypoxia-induced GCN5-mediated acetylation of GLS2 at the K151 site has been shown to enhance the interaction of GLS2 with YAP1

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This was performed in all DOX-containing groups, while control groups received an equivalent volume of saline

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BPC157 was added at concentrations of 5, 10, 20, and 40 g ml-1 for the indicated periods