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pmid:23327997 19

The HGF/c-Met pathway activated by Dihexa promotes neuronal survival through activation of PI3K/Akt signaling cascades that inhibit apoptosis pathways and enhance cellular stress resistance

Kabala, P

Together, these pathways allow neuropeptides like Semax and Selank to reach therapeutically relevant brain concentrations without ever entering systemic circulation in significant amounts

Radioligand and fluorescent binding assays, competition binding, recombinant receptor-expressing cell lines, functional readouts through a defined coupling pathway, and antagonist controls demonstrating that an effect is receptor-mediated