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pylori , but are ineffective to cause lethal oxidative bursts through excitation of endogenous pigments of mammalian cells (Wang et al., 2017)
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(1) Specific involvement of TGR5 in GLP-1 and PYY release is supported by the finding that suppression of TGR5 by siRNA greatly attenuated the increase in cAMP formation and GLP-1 and PYY release in response to OA
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