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Unlike CJC-1295, it lacks the modifications that provide DPP-IV resistance

However, LOsG impaired glucose tolerances (GT), as revealed by OGTT and decreased fast plasma insulin level (Fig

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The observed pathological changes are consistent with aspirins known pharmacological mechanism as a potent inhibitor of platelet aggregation through irreversible acetylation of cyclooxygenase (COX) enzymes, thereby suppressing thromboxane A synthesis