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It appears that the effects on mitochondrial function and inflammatory activity were mediated by E2-dependent increases in Sirt3 expression and mitochondrial protein acetylation (196)
The recognition of a role for GLP-1 in the pathophysiology of congenital and acquired hyperinsulinemic hypoglycemia has brought forward the possibility of targeting the GLP-1 receptor for the treatment of this condition and promising progress has been made to this end
If missing, flag for follow-up because transition-of-coverage evidence can be necessary to assess continuity and avoid improper duplication
When combined, these effects may be more pronounced or persistent
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Receptor activation may modulate immune cell function, cytokine production, and inflammatory signaling pathways independent of metabolic changes
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