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Together, these glucose, pH, enzyme, redox, and field-triggered mechanisms promise logic-gated GLP-1 delivery that synchronizes drug release with metabolic need or lesion biology, maximizing efficacy while limiting off-target exposure

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For example, Mg 2+ -dependent inhibition of K ATP by sulfonylureas, such as tolbutamide and glibenclamide, is evident, suggestive of the importance of Mg 2+ in the impact of sulfonylureas on ion channels, and it may be factored into devising related experiments

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