The autophagy activator rapamycin not only ameliorates fluoride-induced autophagic flux blockage by correcting autophagic degradation impairment but also inhibits fluoride-mediated ferroptosis by reducing the iron content and suppressing lipid peroxidation
Because intravenous therapy delivers glutathione directly into the bloodstream, absorption is immediate
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Enzymatic reactions, such as those involving cytochrome P450, xanthine oxidase, and nitric oxide synthase, can also produce free radicals [14]
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