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+ - Given its potentially adverse effects, ranging from disruption of the insulin system to cancer, the exogenous augmentation of IGF-I is not an attractive or effective method of increasing muscle mass or function
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This paradox may be partially explained by the cellular source: endothelial NOS (eNOS)-derived NO appears protective, while inducible NOS (iNOS) from macrophages tends to promote fibrosis [83]
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