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2.8.3 Data analysis of MPR2 substrate and inhibition study For the substrate assay, the amount of compounds in the vesicle was determined with LC-MS to calculate the transport activity, UR, and inhibition rate using Equations 1113, which were used to determine whether the compounds were substrates of the transporter
Tenuifolin in the prevention of Alzheimers disease-like phenotypes: investigation of the mechanisms from the perspectives of calpain system, ferroptosis, and apoptosis
Mechanisms of cardiovascular remodeling in hyperhomocysteinemia
In their subsequent in vitro studies using cultured proximal tubular cells, they could demonstrate that proximal tubular fluid from nephrotic rats activates IGF-1R, suggesting that filtered IGF-1 is bioactive [218]
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