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According to experiments by Zhu et al., TMAO improved the expression of adenosine diphosphate (ADP) and thrombin to enhance a release of Ca 2+ from intracellular calcium stores, causing platelet aggregation and enhancing thrombosis potential (88)

That work is still in progress, but its implications are important

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And then leveraging the neuroprotective properties of GLP-1 receptor agonists, researchers initiated a Phase II clinical trial to evaluate exenatide therapy for PD