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These processes concertedly impair insulin signaling, -cell function, and inter-organ crosstalk, supporting this axis as the key upstream driver of T2DM progression

(2011) used the histone deacetylase inhibitor depispeptide to reverse DNA hypermethylation and alter the histone modification pattern at GSTP1 promoter, including a reduction of H3K9me2/3 and H3K27me2/3 and an increase of H3K18Ac, thereby inducing GSTP1 mRNA re-expression