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Appetite loss and neurocognitive deficits in late-life depression

These episodes are brief and typically require no intervention

In the central nervous system, GLP-1R signaling within hypothalamic circuits increases satiety and reduces food intake, ultimately promoting body weight reduction

GLP-1-activated PKA inhibits voltage-gated potassium channels, prevents membrane repolarization, and prolongs VDC channel opening to promote Ca 2+ inward currents, contributing to GLP-1 stimulate insulin secretion, and the antagonistic effect of GLP-1R activation on Kv currents in cells requires transactivation of epidermal growth factor receptors, accompanied by signal conduction of cAMP/PKA and phosphatidylinositol-3-kinase (PI3K)/PKC signaling (MacDonald et al., 2003)

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