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Brancaccio, D
Delivery of cells and drugs to the injury site and their long-term maintenance at the site is critical for accelerated target organ repair and functional recovery
Upon genetic deletion of Gclc , MZB differentiation is halted because the GSH-dependent buffering capacity is lost
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The production of free radicals also increases during illness, which can result in complications
Furthermore, our findings could reinforce the previously proven concept that AcM does not hydrolyze PGN of live bacteria, whilst leading to the indirect modulation of the microbiota, driven by the reduced protein availability in the caeca, with some measurable effects on the gene encoding PGN synthesis
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