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NO is an endogenously produced vasodilator and is largely implicated in hemostatic mechanisms [39] while, in tail amputation and heparin-/warfarin-administration models, both the NO-synthase (NOS)-blocker, L-NAME (thought to have a prothrombotic effect) [3] and NOS-substrate, L-arginine [presented with an antithrombotic effect) [3] were little investigated

What may be more within reach is the evaluation of BBB permeability through perfusion studies in AIS patients as a way of avoiding HT as well as including those that would otherwise be ineligible to reperfusion therapies, leaving aside strict and rigid criteria such as time and moving toward a more personalized medicine

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