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This outcome reflects both the inherent difficulty of treating Alzheimer's disease and questions about whether a single neuroprotective mechanism, without addressing amyloid and tau pathology more directly, can be sufficient

We knocked down GDH1 in different liver (Huh7 and Hep3B) and lung (H1299 and H5889) cancer cell lines (Extended Data Fig

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Notably, NFE2 like bZIP transcription factor 2 (NRF2) counters this ferroptosis damage by regulating SLC7A11, offering a potential therapeutic avenue (Sfera et al., 2022)

These risks are uncommon, but they highlight why ongoing medical supervision is important during treatment