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The copper component is biologically essential and tightly regulated research data suggests GHK-Cu actually helps regulate copper metabolism rather than causing copper accumulation
This mechanism is relevant to neuropathy because NF-kB-driven inflammation is a central mediator of nerve damage in diabetic neuropathy, autoimmune neuropathies, and chemotherapy-induced peripheral neuropathy
At 2 mL BAC water and 500 mcg per compound: V draw = 0.5 mg 2 mL10 mg = 0.1 mL = 10 units Payload at 10 units: BPC-157 500 mcg TB-500 500 mcg
However, bioavailability through topical application remains limited compared to injectable delivery
This combined interpretation helps distinguish a primary disturbance in glutathione metabolism from a downstream response to broader cellular stress
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