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The trial enrolled well-characterized T2DM patients presenting with early-stage, objectively verified cognitive impairment

[1] These medications work by mimicking the action of naturally occurring GLP-1, a hormone released from the intestinal tract following food intake

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If liver function is compromised, this conversion becomes less efficient which can further worsen thyroid imbalance

Its unique dual receptor action often results in better glycemic control compared to medications that target just one receptor

This article covers the risks, the scientific mechanism behind these concerns, and how to safely navigate your options