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In order to minimize cellular DNA damage and its consequences, there is a process in the body known as DNA-damage response (DDR)
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In diabetic wound models, BPC-157 was found to promote mature collagen formation in granulation tissue at a level distinct from platelet-derived growth factor (PDGF-BB), underlining its unique mechanistic contribution to the wound healing field
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In a study that investigated the effect of BPC-157 on a rat model of hind limb ischemia, angiogenesis was promoted, helping to restore blood flow to the starved tissues by upregulating VEGFR2, a key regulator of angiogenesis [3]
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