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When they are "broke" (low ATP), they can't afford to fix DNA errors, leading to mutations

In mammals, CHAC1 plays a key role in cell cycle regulation and the maintenance of redox balance and is centrally involved in ferroptosis, stem cell differentiation and development

Aminoglycosides (e.g., gentamicin, amikacin, kanamycin, tobramycin) chelate iron in cochlear tissues, generating ROS that leads to oxidative stress, mitochondrial dysfunction, and programmed cell death

Transferrin receptor 1 (TfR1), a well-known regulator of cellular iron that internalizes the transferrin-bound iron, plays a significant role in ferroptosis, and ROS induction has been shown to enhance TfR1 protein levels and serves as a marker of ferroptosis 8

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