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Ex-4 can significantly rescue memory deficits and neuropathological changes in APP/PS1 AD mice, and it can downregulate the activities of N -acetylglucosaminyltransferase III and bisecting N -acetyl-d-glucosamine through the Akt/GSK-3/-catenin signaling pathway [70]
The early data is compelling enough to take seriously, but its not settled
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