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T cells involved in Con A-induced liver injury are classified as endogenous and exogenous

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Implicated mechanisms include upregulation of molecular pathways involved in oxidative stress, aberrant calcium signaling, silencing of transcription factors and histone deacetylases, and deregulation of cytoskeleton-associated proteins

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Th1 cytokines such as IFN- , IL-2 and TNF- were also increased in the spleen of WT mice injected with Con A, whereas those were not increased in the spleen of Gpx1 KO mice injected with Con A (Figure 5b)