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References: Cingi C, Conk-Dalay M, Cakli H, Bal C

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Intriguingly, the interactions of N-terminal histidine residues of the prion protein (PrP) mainly with Cu 2+ sequestration and accumulation in diseased brain (T 2+ at neutral pH (7.4) ( 2+ is not necessarily pathogenic since it has also been found to produce neuroprotection due to the cis interaction with the C terminal of PrP

Its natural presence in the body contributes to its perceived safety profile

SLC25A39 has also been implicated in colorectal cancer progression by modulating ROS production [36]

"The anti-inflammatory effects of methylsulfonylmethane on lipopolysaccharide-induced inflammatory responses in murine macrophages"