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As described above for PABA/NO, JS-K, and GSNO, the NHGs, in accordance with GT expression patterns, contribute to chemotherapy by producing locally high levels of NO which can rapidly react with O 2 - to generate the potent oxidant peroxynitrite that, in turn causes extensive cellular damage, including the nitration of protein tyrosine residues (Kirsch et al., 2001)

Meanwhile, Sph_12-38 has no significant cytotoxicity to human keratinocytes (HKs) at its high concentration (33.5 M)

In addition, anti-thyroid drugs (e.g

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[4] [6] As an antioxidant, ascorbic acid donates electrons to neutralize reactive oxygen species and free radicals, thereby protecting cellular components from oxidative damage