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Understanding these mechanisms provides the foundation for exploring therapeutic interventions that might support cellular health throughout the aging process

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Consistent with the role of ghrelin signaling in regulating postprandial GLP-1 in lean mice, administration of the ghrelin receptor antagonist d-Lys3 abolished both the increased GLP-1 levels and the reduction in blood glucose levels observed in obese male Tmprss2 / mice following oral glucose challenge (Figure 6H)

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