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Conclusion To summarize, we identified that the decaging-to-ligation strategy can be a powerful tool for tumour-selective ligation between molecules or cells in tubes, cells and living mice

To further explore the underlying mechanism of GSH in activating systemic plant defense responses, we introduced the [Ca 2+ ] cyt reporter GCaMP3 into pad2-1 , generating GCaMP3 / pad2-1 plant, to investigate whether the GSH-deficient mutant attenuated the wound-induced systemic [Ca 2+ ] cyt transmission

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When combined, these peptides are frequently explored in advanced in-vitro and biochemical model systems to better understand coordinated peptide signaling, multi-pathway cellular regulation, and molecular interactions related to tissue-associated research models

NADPH Oxidase Promotes Pancreatic Cancer Cell Survival via Inhibiting JAK2 Dephosphorylation by Tyrosine Phosphatases