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Interestingly, there was no significant difference in the levels of GSH content (0.70 to 0.82 mg/g protein) within the experimental groups (G2, G3, G4) whereas, significantly different from NC and G1 as the former had very high GSH level while the latter had very low levels ( P value = 0.0002)
Given the progressive nature of the disorder, disease pathogenesis is mainly triggered by continual processes such as geometric accumulation of oxidative radicals brought about by cellular inclusions and mitochondrial dysfunction 8,9,10
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