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The mechanism involves altered calcium channel activity, particularly affecting voltage-gated calcium channels (CaVs) and store-operated calcium entry pathways, leading to either excessive calcium influx or impaired calcium release from intracellular stores, ultimately resulting in cellular dysfunction and reduced fertilization potential (39)

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Sources: Rebouche C.J., Seim H., Carnitine Metabolism and Functions, Annual Review of Nutrition (1998)