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It seems as though the amount of GSH depletion that occurs during the CS exposures may trigger the amount of GSH that is maintained in the ELF between exposures
No study has systematically measured the duration of benefit after treatment cessation or established optimal retreatment intervals
doi: 10.18869/nrip.jamsat.3.1.3 38 StadlerDDBaleJFChenardCAReboucheCJ
8 Functions of GSH in redox buffering, detoxification, and drug resistance Glutathione (GSH) is the principal intracellular redox buffer that preserves cellular homeostasis through detoxification of ROS and regeneration of antioxidant capability via GR-catalyzed recycling of its oxidized form (GSSG)
Treatment was very well tolerated
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