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LIGHT signals directly to intestinal epithelia to cause barrier dysfunction via cytoskeletal and endocytic mechanisms

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The proposed mechanism described in papers that were later retracted is that it potentiates hepatocyte growth factor (HGF) signaling at the c-Met receptor, driving dendritic spine formation and synaptogenesis

In addition, in the HFD/STZ-induced SD rat model, Sirt3 overexpression improves painful diabetes neuropathy by activating FoxO3a/PINK1/Parkin-Mediated mitophagy [170]