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The hyper-inflammatory response caused by SARS-CoV-2 induces the process of immunosenescence, increases endothelial cell damage, and leads to myofibrillar degradation and muscle wasting (sarcopenia) due to mitochondrial dysfunction and autophagy [102]

The simultaneous loss of GLP-1R and GIPR signaling leads to increased thymic stromal lymphopoietin (TSLP) release, a higher number of lung ILC2 cells, eosinophils, lymphocytes, and neutrophils in the airway, as well as elevated expression of type 2 cytokines (IL-5 and IL-13), high mobility group box 1 (HMGB1), and intercellular adhesion molecule (ICAM-1) in lung epithelial cells, compared to other mouse strains following exposure to Alternaria extract

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Gross R, Hurwitz R, Marks P

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