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Key Points Studies of the molecular mechanisms of integrin endocytosis support a role for the alternative clathrin adaptor, disabled 2
This is consistent with tolerance of K1A substitution in human Amy (hAmy) for cAMP potency at AMYRs 18
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The compound has attracted scientific interest because of its interaction with nicotinamide N-methyltransferase (NNMT) , an enzyme involved in metabolic processes linked to cellular energy regulation and biochemical signaling pathways
