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The UKPDS trial showed that intensive glucose control in T2D versus conventional treatment (achieving HbA1c of 7.0% versus 7.9%, respectively) reduced overall microvascular complications by 25%
The mechanism of post-prandial increase in GLP-1 level following MBS can be explained by faster gastric emptying and rapid delivery of nutrients to the intestine, exclusion of foregut, and changes in bile acids metabolism [18, 24]
typically improve with gradual dose titration and lifestyle support
New Trends Clin Neuropharmacol 1989;3(1):17-24
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