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Inhibition of p53 disrupts these protective cellular mechanisms, allowing APAP-induced damage to progress unchecked, resulting in severe liver injury
Recent crystallographic studies on PnGGT in complex with GlyGly acceptor demonstrated that the binding mode of GlyGly in the active site is probably the reason for its reduced activity as an acceptor ( Picrophilus torridus GGT (PtGGT), the replacement of an aromatic residue tyrosine at position 327 by asparagine (corresponding residue in EcGGT) introduced significant transpeptidase activity in PtGGT, while the native enzyme exhibited only hydrolytic activity (Rajput et al., 2013)
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