Since the conversion of ADP to ATP requires O 2 as the terminal electron acceptor of electron transport, there is a direct association between ADP concentration and O 2 consumption
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[1] [6] Severe deficiencies may include symptoms of reduced heart function, suppression of bone marrow, as well as more various severe neurological symptoms possibly including sensory loss, abnormal balance and reflexes, memory and consciousness impairment, smell and taste problems, depression, irritability, confusion, and psychosis
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1 H-MRS is a noninvasive approach that enables the determination of in vivo concentrations of specific neurochemicals, including GSH
The combined logic is: Three receptor pathways at once