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The countermeasures are well established: eat enough protein, train with resistance a few times a week, and avoid crash-level deficits

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The pattern is consistent: strong mechanistic plausibility from preclinical data and adjacent human safety, no direct human efficacy or long-term safety data on IGF-1 LR3 itself

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The mechanisms involve upregulating survival genes, reducing inflammatory mediators, activating vascular bypass pathways, and maintaining blood flow despite vessel occlusion

To allow uncoupled respiration, 40 M DNP (2,4-dinitrophenol) was added