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The brain pathology of this disease is similar to that seen in oxidative damage resulting from sulfhydryl depletion due to chronic mercury poisoning (Minamata disease) and is also compatible with decreased cerebral glutathione levels (09)

This allows time for potential recurrence to manifest and be detected

This may result in sense loss, difficulty in proprioception, neuropathic pain, difficulty walking, poor balance, loss of sensation in the feet, muscle weakness, blurred vision (either due to retinopathy [27] or optic neuropathy [28] ), impaired urination, fertility problems, decreased sense of taste and smell, decreased level of consciousness, changes in reflexes, memory loss, mood swings, depression, irritability, cognitive impairment, confusion, anxiety, clumsiness, dementia, and, in more severe cases, psychosis

In relation, Slewa et al

PRMT1 inhibition promotes ferroptosis sensitivity via ACSL1 upregulation in acute myeloid leukemia

Major histocompatibility complex antigen expression in the affected tissues in amyotrophic lateral sclerosis