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The cellular stimulation that produces benefits can also trigger inflammatory responses in reactive skin types

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Germ-free mice (lacking microbiota) show defects in microglial development and an exaggerated neuroinflammatory response, which can be normalized by reintroducing SCFA-producing bacteria [13]

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Sanger sequencing revealed a germline insertion in NF1 exon 2 (c.165_166insCT), causing a frameshift and premature truncation that likely disrupted NF1 function in patient iPSCs (Fig